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Frontiers | Molecular Dynamics of Lipopolysaccharide-Induced Lung Injury in  Rodents | Physiology
Frontiers | Molecular Dynamics of Lipopolysaccharide-Induced Lung Injury in Rodents | Physiology

Fucoidan inhibits LPS-induced acute lung injury in mice through regulating  GSK-3β-Nrf2 signaling pathway | SpringerLink
Fucoidan inhibits LPS-induced acute lung injury in mice through regulating GSK-3β-Nrf2 signaling pathway | SpringerLink

Effects of CH on LPS-induced lung injury in mice. (A) Representative... |  Download Scientific Diagram
Effects of CH on LPS-induced lung injury in mice. (A) Representative... | Download Scientific Diagram

Comparison of direct and indirect models of early induced acute lung injury  | Intensive Care Medicine Experimental | Full Text
Comparison of direct and indirect models of early induced acute lung injury | Intensive Care Medicine Experimental | Full Text

Serum amyloid A promotes LPS clearance and suppresses LPS‐induced  inflammation and tissue injury | EMBO reports
Serum amyloid A promotes LPS clearance and suppresses LPS‐induced inflammation and tissue injury | EMBO reports

Acute Lung Injury Induced by Lipopolysaccharide Is Independent of  Complement Activation | The Journal of Immunology
Acute Lung Injury Induced by Lipopolysaccharide Is Independent of Complement Activation | The Journal of Immunology

Nutrients | Free Full-Text | Attenuation of Lipopolysaccharide-Induced  Acute Lung Injury by Hispolon in Mice, Through Regulating the  TLR4/PI3K/Akt/mTOR and Keap1/Nrf2/HO-1 Pathways, and Suppressing Oxidative  Stress-Mediated ER Stress-Induced Apoptosis ...
Nutrients | Free Full-Text | Attenuation of Lipopolysaccharide-Induced Acute Lung Injury by Hispolon in Mice, Through Regulating the TLR4/PI3K/Akt/mTOR and Keap1/Nrf2/HO-1 Pathways, and Suppressing Oxidative Stress-Mediated ER Stress-Induced Apoptosis ...

Juglanin suppresses fibrosis and inflammation response caused by LPS in  acute lung injury
Juglanin suppresses fibrosis and inflammation response caused by LPS in acute lung injury

Receptor Interacting Protein 3-Mediated Necroptosis Promotes  Lipopolysaccharide-Induced Inflammation and Acute Respiratory Distress  Syndrome in Mice | PLOS ONE
Receptor Interacting Protein 3-Mediated Necroptosis Promotes Lipopolysaccharide-Induced Inflammation and Acute Respiratory Distress Syndrome in Mice | PLOS ONE

JTE-013 Alleviates Inflammatory Injury and Endothelial Dysfunction Induced  by Sepsis In Vivo and In Vitro - Journal of Surgical Research
JTE-013 Alleviates Inflammatory Injury and Endothelial Dysfunction Induced by Sepsis In Vivo and In Vitro - Journal of Surgical Research

Non-digestible oligosaccharides partially prevent the development of LPS-induced  lung emphysema in mice - ScienceDirect
Non-digestible oligosaccharides partially prevent the development of LPS-induced lung emphysema in mice - ScienceDirect

Panaxydol attenuates ferroptosis against LPS-induced acute lung injury in  mice by Keap1-Nrf2/HO-1 pathway | Journal of Translational Medicine | Full  Text
Panaxydol attenuates ferroptosis against LPS-induced acute lung injury in mice by Keap1-Nrf2/HO-1 pathway | Journal of Translational Medicine | Full Text

Carnosic acid protects against lipopolysaccharide‑induced acute lung injury  in mice
Carnosic acid protects against lipopolysaccharide‑induced acute lung injury in mice

Emodin alleviates LPS-induced inflammatory response in lung injury rat by  affecting the function of granulocytes | Journal of Inflammation | Full Text
Emodin alleviates LPS-induced inflammatory response in lung injury rat by affecting the function of granulocytes | Journal of Inflammation | Full Text

IJMS | Free Full-Text | Amelioration of Endotoxin-Induced Acute Lung Injury  and Alveolar Epithelial Cells Apoptosis by Simvastatin Is Associated with  Up-Regulation of Survivin/NF-kB/p65 Pathway | HTML
IJMS | Free Full-Text | Amelioration of Endotoxin-Induced Acute Lung Injury and Alveolar Epithelial Cells Apoptosis by Simvastatin Is Associated with Up-Regulation of Survivin/NF-kB/p65 Pathway | HTML

Figure 5 | Prevention of LPS-Induced Acute Lung Injury in Mice by  Progranulin
Figure 5 | Prevention of LPS-Induced Acute Lung Injury in Mice by Progranulin

Accelerated inflammation and oxidative stress induced by LPS in acute lung  injury: Ιnhibition by ST1926
Accelerated inflammation and oxidative stress induced by LPS in acute lung injury: Ιnhibition by ST1926

Protective effect of oxytocin on LPS-induced acute lung injury in mice |  Scientific Reports
Protective effect of oxytocin on LPS-induced acute lung injury in mice | Scientific Reports

Monitoring lung injury with particle flow rate in LPS‐ and COVID‐19‐induced  ARDS - Stenlo - 2021 - Physiological Reports - Wiley Online Library
Monitoring lung injury with particle flow rate in LPS‐ and COVID‐19‐induced ARDS - Stenlo - 2021 - Physiological Reports - Wiley Online Library

Lung inflammation promotes metastasis through neutrophil protease-mediated  degradation of Tsp-1 | PNAS
Lung inflammation promotes metastasis through neutrophil protease-mediated degradation of Tsp-1 | PNAS

Protective effect of TM6 on LPS-induced acute lung injury in mice |  Scientific Reports
Protective effect of TM6 on LPS-induced acute lung injury in mice | Scientific Reports

Sex-differences in LPS-induced neonatal lung injury | Scientific Reports
Sex-differences in LPS-induced neonatal lung injury | Scientific Reports

Oridonin protects LPS-induced acute lung injury by modulating Nrf2-mediated  oxidative stress and Nrf2-independent NLRP3 and NF-κB pathways | Cell  Communication and Signaling | Full Text
Oridonin protects LPS-induced acute lung injury by modulating Nrf2-mediated oxidative stress and Nrf2-independent NLRP3 and NF-κB pathways | Cell Communication and Signaling | Full Text